EMF Blocking in addition to EMF Shielding
No matter if you live in an apartment or house, or you just would like to ensure your home is free of EMFs There are plenty of ways you can reduce exposure. One of the easiest is to restrict your use of electronic devices. You can also turn to EMF block paint to prevent EMF radiation from entering your house. emf blocking to protect your home against EMF radiations is to use a RF shielding canopy. This is a cloth made of net which contains EMF shielding. It's used to block EMFs from entering a room. Another option is to get your home fitted with a conductive enclosure. These enclosures are known as Faraday cages.
A number of studies have proven that the non-ionizing RF EMF has antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed be based on the downregulation in cancer-related stem cells. This could be the reason for the long-term effects observed in certain patients suffering from advanced HCC. However, the mechanism of AM EMF's impact on cancer patients is not yet clear.
Aspects on the effects of AM RF EMF on HCC tumour growth in vivo were studied in mice. The tumours were divided into 3 groups. First, the group that was unaffected RF EMF. Another group of participants was subjected to RF EMF at the same frequency to the one used by humans. Third group members were exposed to RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on tumors was assessed against the effect of RCF. https://lopez-holden.blogbright.net/info-about-emf-blocking-radiation-1681267930 revealed that tumors treated by HCCMF had significant shrinkage. However, tumours treated with RCF did not show any evidence of tumour shrinkage.
The mechanism behind tumor-specific AM RF EMF might be based on the fact that tumour cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is controlled through CACNA1H which is a protein that mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have wider implications for the treatment and diagnosis of different cancers.
The tumours of the controls were never exposed to EMF from radiofrequency, and fed a normal diet of mice. The tumours in those in the HCCMF group were injected with Huh7 cells after they were 5 to 7 weeks old. The tumors were removed after they had a high burden.
The tumours from the three groups showed different growth curves. The HCCMF-treated tumors had a significant reduction in the size of the tumour after eight weeks. However, the tumours which were treated by RCF did not show any signs of shrinkage. The difference was substantial. The tumors treated by RCF showed necrosis, which is common in tumors that have been exposed to RCF. The possibility is that this necrosis was caused by a lack of oxygen in the larger tumours.
In summary, the results suggest the fact that AM EMF is a powerful source of anticancer activity in vitro and in live. Several studies have shown it is true that AM RF EMF produces measurable tumour shrinkage for HCC patients. The possibility is that the AM EMF produces these effects due to CACNA1H, a protein that is involved in the process of tissue-specific Ca2+ influx. Furthermore, emf blocking may have a long-lasting influence on the growth of HCC tumors in living tissue.